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VITEX for dysmenorrhea
Dysmenorrhea is an extremely common and sometimes debilitating condition for women of reproductive age.
As an inhibitor of prostaglandin synthesis, vitamin E has been studied for dysmenorrhea. It is also suggested that vitamin E has a central analgesic action. This effect might be mediated by a suppressive action on nitric oxide, which has recently been shown to play an important part in central pain processing, or by an inhibitory effect on protein kinase C.
Evidence obtained from randomized controlled trials suggests that vitamin E relieves the pain of primary dysmenorrhea and reduces blood loss.
A more recent study examined the effect of a low dose of vitamin E on dysmenorrhea in 278 young women aged 15 to 17 years. Over 4 months, participants received 400 IU of vitamin E per day or placebo, beginning two days before the expected start of the menstrual period, and continuing through the first three days of bleeding.
Pain was significantly less and of shorter duration among the women in the vitamin E group.
Women in the vitamin E group also lost significantly less blood than did women in the placebo group.
There were no significant side effects associated with vitamin E treatment.
Studies suggest that treatment with VITEX is an effective means of reducing menstrual pain and blood loss among women with primary dysmenorrhea, and maximum effect occurs by about three months.
MED-FOLIC ACID® in cardiovascular diseases
Studies consistently report strong positive associations between total plasma homocysteine and cardiovascular morbidity or mortality or all-cause mortality.
The metabolism of homocysteine and folate are closely linked. Homocysteine is a naturally occurring sulfur-containing amino acid derived from the dietary amino acid methionine.
Raised plasma homocysteine (tHcy) concentrations are caused by genetic mutations, vitamin deficiencies, renal and other diseases, numerous drugs, and increasing age.
Studies have consistently shown a strong relationship with tHcy concentrations and cardiovascular events.
Elevations of plasma tHcy have been linked clinically with CHD, and numerous studies have shown that elevations of homocysteine induce endothelial dysfunction or damage.
Folic acid reduces plasma homocysteine levels and improve insulin and lipid metabolism.
Adequate folate intake is vital because folates are essential for DNA synthesis. Folate status is the most important determinant of an individual’s plasma tHcy concentration and folic acid supplements can safely, effectively, and inexpensively lower total plasma homocysteine concentrations.
Folic acid 5 mg/day improves endothelial function in coronary artery disease.
Endothelial dysfunction or damage is a key early process in atherogenesis.
Endothelial cell dysfunction is emerging as an ultimate culprit for diverse cardiovascular diseases and cardiovascular complications of chronic renal diseases.
Folic acid 5 mg/day significantly enhances endothelial function and reduces blood pressure in chronic smokers.
Cigarette smoking is associated with increased plasma homocysteine concentrations, endothelial dysfunction and arterial stiffening.
Folic acid, a simple, safe and inexpensive vitamin intervention is useful in primary cardiovascular prevention in this high-risk group.
Folic acid supplementation also improves endothelial function and lipid profile in post-menopausal women.
High dose folic acid reverses endothelial dysfunction in patients with diabetes and hypercholesterolemia.
Patients with diabetes are characterized by endothelial dysfunction and cardiovascular mortality. Folic acid can be used to improve nitric oxide status and to restore endothelial dysfunction in patients with type II diabetes.
Supplementation with folic acid (5–10 mg/day) restores impaired endothelium dependent vasodilatation in patients with hyperhomocysteinemia, diabetes or hypercholesterolemia
Product of the month: FOLAC
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Frequently Asked Questions
CLAMYCIN®
FONGINOX®
OFLOMED®
OMEPRAL®
CLAMYCIN®
Q: Why CLAMYCIN® is indicated for ulcer
disease?
A: CLAMYCIN® contains Clarithromycin, a macrolide
antibiotic that acts against most strains of Helicobacter pylori, the bacteria
involved in the pathogenesis of gastro-duodenal ulcers.
Q: How to take CLAMYCIN® for ulcer disease?
A: CLAMYCIN® is given 2 tablets 2 times daily as triple therapy in
association with anti-ulcer and other ant-infective drugs for 14 days.
FONGINOX®
Q: What is the pulse therapy for the treatment by FONGINOX®?
A: The pulse therapy is an unique dosing regimen in onychomycosis
therapy that consists of a one-week pulse of 200 mg (2 capsules
of FONGINOX®) twice daily, then a three week rest period.
Two FONGINOX® pulses are required for fingernail fungus and
three for toenail fungus.
Q: What are the advantages of using the pulse therapy?
A: The pulse therapy makes you exposed to less drug, the treatment time is
shorter and the cost of the treatment is greatly reduced.
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OFLOMED®
Q: How to take OFLOMED®?
A: Take OFLOMED®
exactly as prescribed by your doctor. The daily dose is usually 1 to 2 tablets
a day. A daily dose up to 2 tablets may be taken as a single dose preferably in
the morning.
OFLOMED® can be taken on an empty stomach or with
meals.
Q: How long to take OFLOMED®?
A: The duration
of treatment is determined according to the disease process. The duration
should not exceed 2 months.
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OMEPRAL®
Q: Can we use OMEPRAL® for the treatment of
gastroesophageal reflux?
A: Yes. OMEPRAL® has shown an extensively
researched and proven efficacy in the treatment of gastroesophageal reflux,
leading to a rapid relief of the symptoms. Its safety and tolerability were
widely confirmed.
Q: How to take OMEPRAL® for the treatment of
gastroesophageal reflux?
A: Take OMEPRAL® exactly as prescribed by
your doctor.
The daily dose is 1 capsule usually taken in the morning for 4
to 8 weeks.
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